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Ace the American Board of Pediatrics Exam with Med-Challenger's proven Pediatrics Board Review for ABP exam preparation. Featuring over 2,100 Pediatrics Board Exam Review questions with detailed explanations and rationales following the ABP Exam Blueprint, board exam simulation, adaptive learning features, and hundreds of CME credits, Med-Challenger's pediatrics exam review is the best way to prepare for the pediatrics board exam.

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Pediatrics Course Information

Pediatrics Review Course Information

Growth and Development

Development Milestones
Failure to Thrive
Normal Growth
Obesity

Preventive Pediatrics

Anticipatory Guidance
Childhood Immunizations
Disease Prevention
Periodic Health Examination / Screening

Poisoning and Environmental Exposure to Hazardous Substances

Acetaminophen Toxicity
Anticholinergics Toxicity
Anticonvulsant Toxicity
Antidepressants Toxicity (incl. Serotonin Syndrome)
Aspirin and NSAIDs Toxicity
Cardiovascular Drug Toxicity
Caustic Ingestion
Cocaine and Other Sympathomimetics Toxicity
Hallucinogen Toxicity
Heavy Metal Toxicity
Hydrocarbon Toxicity
Inhaled Toxins
Lithium Toxicity
Opiate Toxicity
Pesticide Toxicity (incl. Organophosphates)
Sedatives and Hypnotics Toxicity (incl. Barbiturates, Benzodiazepines)
Toxic Alcohols
Toxic Plant Ingestions (incl. Mushrooms)

Fetus and Newborn Infant

Delivery Room
Effects of Maternal Conditions, Medications, and Substance Abuse
Jaundice in Newborn Infants
Newborn Conditions and Diseases
Newborn Respiratory Distress and Cyanosis
Routine Care of the Newborn Infant

Genetics and Dysmorphology

Inheritance
Gene Syndromes and Associations
Diagnostic Testing and Counseling
Chromosome Abnormalities
Gene Abnormalities

Allergic and Immunologic Disorders

Allergic Reactions
Allergic Rhinitis
Allergy-Induced Asthma
Immunodeficiency Disorders
Allergy Testing & Immunotherapy

Infectious Diseases

Bacterial Infections
HIV/AIDS
Infections in Immunocompromised Hosts
MRSA in the Pediatric Population
Parasitic Infections
Prevention of Infectious Disease
Soft Tissue Infections
Systemic Fungal Infections
Viral Infections

Endocrinology, Metabolism, and Nutrition

Adrenal Insufficiency
Diabetes
Disorders of Calcium
Metabolism Fluids and Electrolytes
Hypoglycemia
Inborn Errors of Metabolism
Infant Feeding
Lactic Acidosis
Normal Nutritional Requirements
Thyroid Disease

Gastrointestinal Disorders

Acute and Chronic Abdominal Pain
Constipation
Diarrhea
Esophageal Disorders and Foreign Bodies
Gastrointestinal Bleeding
Gastrointestinal Disorders in Newborn Infants
Inflammatory Bowel Disease
Jaundice
Malabsorption
Vomiting

Respiratory Disorders

Parenchyma
Asthma
Extrapulmonary / Heart Failure
Infant and Ciliary Conditions
Lower Airway
Pneumonia
Signs and Symptoms
Sleep Apnea / Respiratory Diagnostic Testing
Upper Airway SIDS and Brief Resolved Unexplained Events (BRUE)

Cardiovascular Disorders

Clinical Presentations of Cardiovascular Disorders
Congestive Heart Failure
Congenital Heart Disease
Acquired Heart Disease
Myocarditis and Pericarditis
Rate and Rhythm Disorders
Systemic Diseases Affecting the Heart
Cardiomyopathies
Hypertension
Valvular Heart Disease

Blood and Neoplastic Disorders

Erythrocyte Disorders
Coagulation Disorders
Leukocyte Disorders
General Aspects of Blood and Neoplastic Disorders
Neoplastic Disorders
Platelet Disorders
Pancytopenia

Renal and Urologic Disorders

General Renal and Urologic Disorders
Infections of the Urinary Tract
Renal Failure

Neurologic Disorders

Altered Levels of Consciousness and Neurodegenerative Disorders
Cerebrovascular Disease
Headaches & Intracranial Pressure
Infectious Causes of Neurologic Disorders
Movement Disorders (incl. Ataxia)
Peripheral Nerve and Neuromuscular Junction Disorders
Seizures
Spinal Cord Disease and Dysraphism
Weakness and Muscle Disease (incl. Hypotonia and Cerebral Palsy)

Musculoskeletal Disorders, Orthopedics, and Sports Medicine

Evaluation for Sports Participation
Musculoskeletal Disorders
Orthopedic Injuries

Skin Disorders

Acne
Dermatitis
Infectious Rashes
Other Dermatologic Disorders
Pigmented Lesions
Skin Disorders in Newborn Infants

Collagen Vascular and Other Multisystem Disorders

Arthritis
Systemic Lupus Erythematosus
Vasculitis Syndromes

Eye, Ear, Nose and Throat Disorders

Dental and Oral Disorders
EENT Infections
External Disorders of the Eye
Internal Disorders of the Eye
Miscellaneous Disorders of the Eye
Nasal Disorders
Throat Disorders

Adolescent Medicine and Gynecology

Female Pubertal Development
Gynecologic Health Care
Male Pubertal Development
Pregnancy and Contraception
Self-Identity and Family Influence
Sexuality and Emotional Development
Sexually Transmitted Infections

Behavioral and Mental Health Issues

Autism Spectrum Disorders
Child Abuse
Disorders of Attention and Impulse Control
Eating Disorders
Mood and Affect Disorders
Suicidal Behavior, Psychotic Behavior, Thought Disorders
Pediatric Substance Abuse

Critical and Emergency Care

Abdominal, Chest, and Multisystem Trauma
Burns and Fractures
Common Conditions Requiring Life Support
Emergency Life Support
Emergent Fever and Seizure Care
Head and CNS Trauma
Impending Systemic Failure
Wounds

Pharmacology and Pain Management

ADHD: Non-Stimulant Medications
ADHD: Stimulant Medications
Pain Management
Pharmacology
Procedural Sedation

Ethics, Patient Safety, and Quality Improvement

Communication, Family, and Cultural Issues
End-of-Life Care
Medical Error Prevention
Medical Ethics

Sample Question 1

A 17-year-old presents to you with an acute case of cocaine ingestion.

Which of the following is true?

Neurologic complaints after cocaine use are rare.
Cocaine-induced seizures are typically focal in nature.
Cocaine-induced strokes are universally hemorrhagic.
Cardiopulmonary complaints are most common complication after cocaine use.

Cocaine has a diffuse physiologic influence. Cardiopulmonary adverse events following cocaine use are the most common; chest pain was reported by 40% of patients with recent cocaine use presenting to emergency departments. Neurologic symptoms (39.1%) are the second most common cause of cocaine-related complaints.

Cocaine-induced strokes may be either ischemic or hemorrhagic and may be secondary to powdered cocaine (hydrochloride) and crack (alkaloids).

Chronic cocaine use causes structural abnormalities that may result in reentrant dysrhythmias and other conduction disturbances.

Acute coronary artery vasoconstriction after cocaine use is mediated through alpha-adrenergic stimulation.

Cocaine-induced seizures are usually generalized with tonic–clonic features and are reported to occur within minutes (intravenous) to 12 hours following cocaine use.

Reference:

Dittmar PK, Olmedo R. An evidence-based approach to cocaine-associated emergencies. Emerg Med Pract. 2008;10(1):1-20. Accessed June 1, 2015.

Sample Question 2

Antibiotic treatment of streptococcal pharyngitis or tonsillitis not only shortens the duration of symptoms, but also prevents some complications, such as rheumatic fever. Not all serious streptococcal infections, however, begin with pharyngitis. Which of the following diseases is least likely to be associated with a recent history of streptococcal pharyngitis?

peritonsillar abscess
streptococcal lymphadenitis
tonsillopharyngeal cellulitis
invasive group A strep disease
retropharyngeal abscess

Invasive group A strep causes rapidly progressive disease, usually manifested as a toxin-mediated necrotizing soft tissue infection. Only about 5% of cases of invasive group A strep disease are preceded by symptomatic pharyngitis; breaches in the skin more readily create a portal of entry. The remaining complications result from extension of infection to the tonsils, retropharyngeal space, and lymph nodes. Otitis media and sinusitis also occur commonly following streptococcal pharyngitis.

Sample Question 3

Choose the condition that is the most common cause of upper gastrointestinal hemorrhage:

esophageal varices
erosive gastritis or esophagitis
peptic ulcer disease
arteriovenous malformations, tumors, and aortoenteric fistulas (after grafting)
Mallory-Weiss tear

Peptic ulcer disease is the most common cause of upper GI hemorrhage, representing about 50% of cases, half of which are duodenal in origin. Gastritis/esophagitis accounts for 20% and varices for 12%. Mallory-Weiss tears account for only about 10% of cases overall, and only occasional cases have been reported in children. When Mallory-Weiss tears occur, there is usually a history of antecedent retching, vomiting, hiccoughing, or paroxysmal coughing, but in nearly half of cases no such history is obtained.

Sample Question 4

An infant boy with a gestational age of 29 weeks has bounding peripheral arterial pulses and a wide pulse pressure. He develops respiratory distress and tachypnea. The most common cause of congestive heart failure (CHF) in the premature neonate is which of the following?

coarctation of the aorta
severe anemia
hypoplastic left ventricle
patent ductus arteriosus (PDA)

Educational Objective:

Identify the most common cause of congestive heart failure in a premature infant.

Key Point:

PDA is the most common cause of CHF in a premature infant. The smooth muscle in the PDA is less likely to constrict and close.

Explanation:

In utero, most pulmonary arterial blood shunts right to left through the ductus arteriosus into the aorta. Because the pulmonary vascular resistance decreases after birth, the shunt through the arteriosus reverses (becoming left to right). The high arterial PO2 constricts and closes the ductus.

PDA is a common condition in premature infants because the smooth muscle in the wall of the ductus is less responsive to high PO2 and less likely to constrict after birth. A premature infant with a large PDA is at high risk for the development of pulmonary vascular disease and CHF.

References:

Bernstein D. Heart failure (chapter 442). In: Kliegman RM, et al, eds. Nelson Textbook of Pediatrics. 20th ed., 2016:2282-228.

Kliegman RM, et al. Acyanotic congenital heart disease: left-to-right shunt lesions (chapter 426). In: Kliegman RM, et al, eds. Nelson Textbook of Pediatrics. 20th ed., 2016:2189-2199.

Sample Question 5

A 19-year-old girl presents to you for a sports physical examination. You discover that she has splenomegaly. Urine dipstick analysis is positive for red blood cells (RBCs), but microscopic urinalysis shows no RBCs.

She is found to have normocytic anemia with a hemoglobin level of 8.6 g/dL. She has a reticulocyte count of 5% and a low-normal ferritin level. Her lactate dehydrogenase level is elevated, as is her indirect bilirubin level. Her haptoglobin levels are low. Her family history is significant for gallbladder disease.

A Coombs test is performed and is negative. Hemoglobin electrophoresis is negative. Peripheral blood smear shows many rounded RBCs approximately two-thirds of their normal size.

Which of the following tests should be ordered to confirm your suspicions?

repeat Coombs test
chromosome analysis
sickle cell preparation
osmotic fragility testing
glucose-6-phosphate dehydrogenase (G6PD) levels

Educational Objective:

Recognize clinical and laboratory findings associated with hereditary spherocytosis.

Key Point: 

The diagnosis of hereditary spherocytosis is based on clinical findings, spherocytes, and positive osmotic fragility testing.

Explanation:

Osmotic fragility testing should be ordered. Hereditary spherocytosis is an autosomal dominant disorder that causes a weakened RBC membrane skeleton. Anemia, jaundice, and splenomegaly are the most common clinical features of hereditary spherocytosis. The degree of anemia is extremely variable and may be absent, mild, moderate, or severe. The spherocytes are hemolyzed in the spleen. The condition is distinguished from autoimmune spherocytosis by the Coombs test. The diagnosis is made based on the clinical presentation, presence of spherocytes, and a positive screening test such as osmotic fragility testing.

Although splenectomy can cure chronic hemolytic anemia due to hereditary spherocytosis, it is usually delayed as long as possible due to considerations for risk of sepsis, particularly in children. Due to the chronic bilirubinemia, patients with hereditary spherocytosis often have gallstones.

References:

Christensen RD, Yaish HM, Gallagher PG. A pediatrician's practical guide to diagnosing and treating hereditary spherocytosis in neonates.  Pediatrics. 2015;135(6):1107-1114.

Luzatto L. Hemolytic anemias and anemia due to acute blood loss. In: Longo D, et al., eds. Harrison’s Principles of Internal Medicine. 18th ed., 2011:872-886.

Target Audience

Med-Challenger PEDS is intended for practitioners of acute and primary care or clinicians otherwise involved in the diagnosis and treatment of acute, emergent, or chronic medical problems.

Educational Objectives

After completing this activity, the participant should be better able to:

Manage the broad spectrum of acute and chronic medical problems encountered in the practice of pediatric medicine.
Formulate appropriate differential diagnoses for patients presenting with acute and chronic medical problems of uncertain etiology.
initiate therapeutic and diagnostic procedures for patients presenting with acute and chronic medical disorders.
Manage the initial treatment of acute and chronic disorders effectively.
Diagnose and treat the spectrum of common pediatric disorders involving all organ systems.
Demonstrate mastery of the knowledge and cognitive skills required to achieve objectives 1-5 by scoring at least 80% on all topics studied for CME credit.

Faculty

Kathleen K. Aiello, MD, FAAP
Pediatrician
Ambler Pediatrics
Ambler, PA

Joint Accreditation Statement

In support of improving patient care, this activity has been planned and implemented by the Postgraduate Institute for Medicine and Challenger Corporation. Postgraduate Institute for Medicine is jointly accredited by the Accreditation Council for Continuing Medical Education (ACCME), the Accreditation Council for Pharmacy Education (ACPE), and the American Nurses Credentialing Center (ANCC), to provide continuing education for the healthcare team.

Designation Statement

AMA Credit - The Postgraduate Institute for Medicine designates this enduring material for a maximum of 167.0 AMA PRA Category 1 Credit(s)™. Physicians should claim only the credit commensurate with the extent of their participation in the activity.

ANCC Credit - The maximum number of hours awarded for this Continuing Nursing Education activity is 167.0 contact hours.

ABP MOC Points - Successful completion of this CME activity, which includes participation in the activity and individual assessment of and feedback to the learner, enables the learner to earn up to 167 MOC points in the American Board of Pediatrics’ (ABP) Maintenance of Certification (MOC) program. It is the CME activity provider’s responsibility to submit learner completion information to ACCME for the purpose of granting ABP MOC credit.

AAP Credit - This continuing medical education activity has been reviewed by the American Academy of Pediatrics and is acceptable for a maximum of 167.00 AAP credits. These credits can be applied toward the AAP CME/CPD Award available to Fellows and Candidate Members of the American Academy of Pediatrics.

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Pediatrics Board Review
Product Comparison

Take a look at premiere Pediatrics knowledge products from leading vendors.

FEATURE	MED-C PEDS	Pass Machine	True Learn	Board Vitals
Questions	1700+	600	1000	1500
AMA Cat 1 CME	167	54.75	No CME	45
AAP CME	167	0	No CME	0
ABP MOC Part 2 Points	167	No Points	No Points	30
Ongoing Updates	Yes	No	No	No
Exam Simulator	Yes	No	No	No
Learning Rx	Yes	No	No	No
Low-Cost Renewal	Yes	No	No	No

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